Inden for emnet celleskade beskrives der i Robbins en lang række molekylære signaleringsveje og andre molekylære forhold. Skal vi kende til og kunne beskrive fx dannelsen af ROS i leukocytter og den enzymatiske elimination af ROS?
Added by: Karen
Inden for emnet celleskade beskrives der i Robbins en lang række molekylære signaleringsveje og andre molekylære forhold. Skal vi kende til og kunne beskrive fx dannelsen af ROS i leukocytter og den enzymatiske elimination af ROS?
Added by: Karen
When going through old examinations in basal pathology, 5th semester medicine, I wondered how, on one hand, dysplasia is defined as being a reversible, non-neoplastic condition, but on the other hand, the term “dysplastic” is used to describe cytologic and histologic changes in benign and malignant tumours. Can you explain this?
I’m sorry but what about hemophilia type A? What is the difference between its pathogenesis and etiology? I’m getting confused because it is an inherited disease. So, the etiology is gene disorder, right?
Dear teachers (and students) of pathology,
I am having some trouble distinguishing between etiology and pathogenesis.
E.g. during atherosclerosis:
– Etiology = e.g. cholesterol, hypertension, smoking, obesity, diabetes etc.
– Pathogenesis = intimal inflammation (possibly caused by the deposition of cholesterol).
Is it “simply” a question of cause and effect/process?
Added by: Sara
On: May 8th 2012